By Aneesh B. Singhal, Eng H. Lo, Turgay Dalkara, Michael A. Moskowitz (auth.), R. Gilberto González, Joshua A. Hirsch, Michael H. Lev, Pamela W. Schaefer, Lee H. Schwamm (eds.)
This up-to-date moment version of Acute Ischemic Stroke: Imaging and Intervention presents a accomplished account of the cutting-edge within the analysis and remedy of acute ischemic stroke. the elemental structure of the 1st version has been retained, with sections on basics comparable to pathophysiology and explanations, imaging innovations and interventions. besides the fact that, each one bankruptcy has been revised to mirror the $64000 fresh growth in complex neuroimaging and using interventional instruments. additionally, new chapters are integrated at the scientific ischemic penumubra and using complicated neuroimaging to pick sufferers for neurointerventional systems. all the authors are the world over famous specialists and individuals of the interdisciplinary stroke group on the Massachusetts basic medical institution and Harvard clinical college. The textual content is supported by means of various informative illustrations, and simplicity of reference is ensured in the course of the inclusion of appropriate tables. This publication will function a distinct resource of updated info for neurologists, emergency physicians, radiologists and different health and wellbeing care services who deal with the sufferer with acute ischemic stroke.
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Additional info for Acute Ischemic Stroke: Imaging and Intervention
Zivin JA, Mazzarella V. Tissue plasminogen activator plus glutamate antagonist improves outcome after embolic stroke. Arch Neurol. Andersen M, Overgaard K, Meden P, Boysen G, Choi SC. Effects of citicoline combined with thrombolytic therapy in a rat embolic stroke model. Stroke. Yang Y, Li Q, Shuaib A. Enhanced neuroprotection and reduced hemorrhagic incidence in focal cerebral ischemia of rat by low dose combination therapy of urokinase and topiramate. Neuropharmacology. Bowes MP, Rothlein R, Fagan SC, Zivin JA.
Embolic Infarctions Carotid stenosis can also cause low-flow stroke when the collateral flow from the anterior communicating artery (ACoA), PCoA, and retrograde through the ophthalmic artery is insufficient to perfuse the ipsilateral hemisphere. Low flow causes symptoms and infarction in the distal cortical borderzone territory between the distal branches of the ACA, MCA, and PCA . The actual boundaries between these territories may shift due to increased flow through the ACA or PCA to supply the MCA.
The brain is such an exquisitely sensitive reporting system that small infarctions, well below the size that causes clinical signs in other organ systems, can cause major disability in the brain. 1) . 2, including atherosclerotic, cardiogenic, and lacunar (penetrating vessel) mechanisms. Large series have failed to identify a definite cause in 25–39% of patients with ischemic stroke, depending on the quality, completeness, and quickness of the clinical workup . This group of strokes of unknown causes is known as cryptogenic stroke.
Acute Ischemic Stroke: Imaging and Intervention by Aneesh B. Singhal, Eng H. Lo, Turgay Dalkara, Michael A. Moskowitz (auth.), R. Gilberto González, Joshua A. Hirsch, Michael H. Lev, Pamela W. Schaefer, Lee H. Schwamm (eds.)